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Inflammation and lung cancer[electro...
~
Dubinett, Steven M.
Inflammation and lung cancer[electronic resource] /
紀錄類型:
書目-語言資料,印刷品 : Monograph/item
杜威分類號:
616.99424
書名/作者:
Inflammation and lung cancer/ edited by Steven M. Dubinett.
其他作者:
Dubinett, Steven M.
出版者:
New York, NY : : Springer New York :, 2015.
面頁冊數:
x, 212 p. : : ill., digital ;; 24 cm.
Contained By:
Springer eBooks
標題:
Lungs - Cancer
標題:
Lungs - Cancer
標題:
Inflammation.
標題:
Biomedicine.
標題:
Cancer Research.
標題:
Pathology.
標題:
Medical Microbiology.
標題:
Immunology.
ISBN:
9781493927241 (electronic bk.)
ISBN:
9781493927234 (paper)
內容註:
Inflammation and Lung Cancer: The relationship to COPD -- Inflammation and Lung Cancer: The role of EMT -- Inflammation and Lung Cancer: Molecular pathology -- Inflammation and Lung Cancer: Prevention -- Inflammation and Lung Cancer: The Link to Angiogenesis -- Inflammation and Lung Cancer: Eicosanoids -- Inflammation and Lung Cancer: Addressing Inflammation with Immunotherapy.
摘要、提要註:
This volume presents aspects of the inflammatory tumor microenvironment (TME) and its many roles in tumor progression and metastasis, including creation of a hypoxic environment, increased angiogenesis and invasion, changes in expression of microRNAs (miRNAs), and an increase in a stem cell phenotype. The volume also covers the mechanisms of inflammatory mediators. Chronic overexpression of inflammatory mediators in the TME, as seen in smokers and patients with non-small cell lung cancer (NSCLC), can also lead to increased tumor initiation, progression, invasion, and metastasis. The volume will provide a comprehensive perspective of the latest findings and summaries of progress made regarding inflammation and its connection to lung cancer.
電子資源:
http://dx.doi.org/10.1007/978-1-4939-2724-1
Inflammation and lung cancer[electronic resource] /
Inflammation and lung cancer
[electronic resource] /edited by Steven M. Dubinett. - New York, NY :Springer New York :2015. - x, 212 p. :ill., digital ;24 cm.
Inflammation and Lung Cancer: The relationship to COPD -- Inflammation and Lung Cancer: The role of EMT -- Inflammation and Lung Cancer: Molecular pathology -- Inflammation and Lung Cancer: Prevention -- Inflammation and Lung Cancer: The Link to Angiogenesis -- Inflammation and Lung Cancer: Eicosanoids -- Inflammation and Lung Cancer: Addressing Inflammation with Immunotherapy.
This volume presents aspects of the inflammatory tumor microenvironment (TME) and its many roles in tumor progression and metastasis, including creation of a hypoxic environment, increased angiogenesis and invasion, changes in expression of microRNAs (miRNAs), and an increase in a stem cell phenotype. The volume also covers the mechanisms of inflammatory mediators. Chronic overexpression of inflammatory mediators in the TME, as seen in smokers and patients with non-small cell lung cancer (NSCLC), can also lead to increased tumor initiation, progression, invasion, and metastasis. The volume will provide a comprehensive perspective of the latest findings and summaries of progress made regarding inflammation and its connection to lung cancer.
ISBN: 9781493927241 (electronic bk.)
Standard No.: 10.1007/978-1-4939-2724-1doiSubjects--Topical Terms:
626770
Lungs
--Cancer
LC Class. No.: RC280.L8
Dewey Class. No.: 616.99424
Inflammation and lung cancer[electronic resource] /
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This volume presents aspects of the inflammatory tumor microenvironment (TME) and its many roles in tumor progression and metastasis, including creation of a hypoxic environment, increased angiogenesis and invasion, changes in expression of microRNAs (miRNAs), and an increase in a stem cell phenotype. The volume also covers the mechanisms of inflammatory mediators. Chronic overexpression of inflammatory mediators in the TME, as seen in smokers and patients with non-small cell lung cancer (NSCLC), can also lead to increased tumor initiation, progression, invasion, and metastasis. The volume will provide a comprehensive perspective of the latest findings and summaries of progress made regarding inflammation and its connection to lung cancer.
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