大葉大學圖書館 |

Systemic sclerosis[electronic resource] /

紀錄類型:	書目-語言資料,印刷品 : Monograph/item
杜威分類號:	616.77
書名/作者:	Systemic sclerosis/ edited by Kazuhiko Takehara, Manabu Fujimoto, Masataka Kuwana.
其他作者:	Takehara, Kazuhiko.
出版者:	Tokyo : : Springer Japan :, 2016.
面頁冊數:	viii, 247 p. : : ill., digital ;; 24 cm.
Contained By:	Springer eBooks
標題:	Systemic scleroderma.
標題:	Systemic scleroderma - Research.
標題:	Medicine & Public Health.
標題:	Dermatology.
標題:	Rheumatology.
標題:	Internal Medicine.
ISBN:	9784431557081
ISBN:	9784431557074
內容註:	1 -- Fibrosis: overview 2 -- Autoimmunity in systemic sclerosis: Overview 3 -- Endothelial progenitor cell 4 -- Animal models of systemic sclerosis 5 -- Genetics of Systemic Sclerosis 6 -- Role of the Interleukin-1 Family in the Fibrogenic Phenotype in Systemic Sclerosis 7 -- Interleukin-6 in the Pathogenesis of Systemic Sclerosis 8 -- Epigenetics 9 -- Transforming Growth Factor-ß and Connective Tissue Growth Factor 10 -- Endothelin 11 -- The Role of B cells in Systemic Sclerosis 12 -- Fli1 13 -- Biomarker 14 -- Autoantibodies in Systemic Sclerosis.
摘要、提要註:	This book provides a comprehensive review of the recent advances in the research of systemic sclerosis (scleroderma) Systemic sclerosis is a complex autoimmune disease characterized by vasculopathy, fibrosis, and immune activation. This disease is also associated with certain susceptibility genes and is affected by epigenetic regulation. Recent studies have revealed a variety of new evidence suggestive of the pathogenesis of the disease. These include endothelial progenitor cells, cytokines and growth factors, and B cells. Studies using animal models have also provided novel insight into the pathomechanisms. In addition to these topics, the book covers clinical research regarding biomarkers and autoantibodies. This volume will benefit all rheumatologists and dermatologists as well as basic scientists, especially immunologists, molecular biologists, and biochemists.
電子資源:	http://dx.doi.org/10.1007/978-4-431-55708-1

Systemic sclerosis[electronic resource] /
Systemic sclerosis[electronic resource] /edited by Kazuhiko Takehara, Manabu Fujimoto, Masataka Kuwana. - Tokyo :Springer Japan :2016. - viii, 247 p. :ill., digital ;24 cm.

1 -- Fibrosis: overview 2 -- Autoimmunity in systemic sclerosis: Overview 3 -- Endothelial progenitor cell 4 -- Animal models of systemic sclerosis 5 -- Genetics of Systemic Sclerosis 6 -- Role of the Interleukin-1 Family in the Fibrogenic Phenotype in Systemic Sclerosis 7 -- Interleukin-6 in the Pathogenesis of Systemic Sclerosis 8 -- Epigenetics 9 -- Transforming Growth Factor-ß and Connective Tissue Growth Factor 10 -- Endothelin 11 -- The Role of B cells in Systemic Sclerosis 12 -- Fli1 13 -- Biomarker 14 -- Autoantibodies in Systemic Sclerosis.

This book provides a comprehensive review of the recent advances in the research of systemic sclerosis (scleroderma) Systemic sclerosis is a complex autoimmune disease characterized by vasculopathy, fibrosis, and immune activation. This disease is also associated with certain susceptibility genes and is affected by epigenetic regulation. Recent studies have revealed a variety of new evidence suggestive of the pathogenesis of the disease. These include endothelial progenitor cells, cytokines and growth factors, and B cells. Studies using animal models have also provided novel insight into the pathomechanisms. In addition to these topics, the book covers clinical research regarding biomarkers and autoantibodies. This volume will benefit all rheumatologists and dermatologists as well as basic scientists, especially immunologists, molecular biologists, and biochemists.

ISBN: 9784431557081

Standard No.: 10.1007/978-4-431-55708-1doiSubjects--Topical Terms:

468500
Systemic scleroderma.

LC Class. No.: RC924.5.S34

Dewey Class. No.: 616.77

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520 $a This book provides a comprehensive review of the recent advances in the research of systemic sclerosis (scleroderma) Systemic sclerosis is a complex autoimmune disease characterized by vasculopathy, fibrosis, and immune activation. This disease is also associated with certain susceptibility genes and is affected by epigenetic regulation. Recent studies have revealed a variety of new evidence suggestive of the pathogenesis of the disease. These include endothelial progenitor cells, cytokines and growth factors, and B cells. Studies using animal models have also provided novel insight into the pathomechanisms. In addition to these topics, the book covers clinical research regarding biomarkers and autoantibodies. This volume will benefit all rheumatologists and dermatologists as well as basic scientists, especially immunologists, molecular biologists, and biochemists.
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